Vasculitis
Jul 15, 2024 15:06:53 GMT
Post by Admin on Jul 15, 2024 15:06:53 GMT
Someone on FB asked about their headaches;
“Every time I've tried any treatment I have worsened head pain, where my temples feel extremely sore/bruised, aching, tight constriction in every tendon or blood vessel, nerve artery, swelling protruding veins, sharp pain and its absolutely unbearable.”
I responded by saying this;
“Bartonella causes vasculitis in the whole body and when it's around the temples it's called Temporal Arteritis . There are a lot of different treatments that will affect it and when that happens the release of die off toxins cause inflammation there. The only treatment that does a complete job is the immune system but you need to turn that back on to get it to work. You also need to fix your detoxing ability to make the experience less of a burden.”
my.clevelandclinic.org/health/diseases/temporal-arteritis-giant-cell-arteritis
“Temporal arteritis, or giant cell arteritis, is a kind of vasculitis, which is inflammation of your blood vessels. With this type of vasculitis, inflammation causes your temporal arteries to swell and narrow, which restricts blood flow. The most common symptom of temporal arteritis is a throbbing headache.”
Antineutrophil cytoplasmic antibody or ANCA is an antibody that are primarily IgG. They are a type of antibody that can attach to neutrophils and cause them to attack blood vessels, which can lead to vasculitis.
This is considered an autoimmune disease but what are the neutrophils actually attacking?
pubmed.ncbi.nlm.nih.gov/34500468/
“Bartonella and Coxiella infections presenting as systemic vasculitis”
“Six cases of Bartonella infections were added to 18 cases from literature review. Causative pathogens were mainly B. henselae. Bartonella infection mimicked ANCA-associated vasculitis in 83%”
www.ncbi.nlm.nih.gov/pmc/articles/PMC6120290/
“Bartonella Endocarditis Mimicking Crescentic Glomerulonephritis with PR3-ANCA Positivity”
Obviously the heart is part of the vascular system.
pubmed.ncbi.nlm.nih.gov/38222325/
“Bartonella henselae infection-mediated shunt nephritis”
“Renal biopsy showed membranoproliferative glomerulonephritis with full-house immunofluorescence pattern. Low serum complements, PR3 ANCA positivity and elevated Bartonella immunoglobulin titers suggested a diagnosis of infective endocarditis-associated glomerulonephritis.”
Nephritis is Kidney disease.
“Is It ANCA Vasculitis or Bartonella”
When treating a Bartonella infection it might be easier to understand if you think of it as killing infected cells causing vasculitis.
pubmed.ncbi.nlm.nih.gov/11553014/#
“Evidence of a leading role for VEGF in Bartonella henselae-induced endothelial cell proliferations”
“In fact, B. henselae induces host cell production of the angiogenic factor vascular endothelial growth factor (VEGF), leading to proliferation of endothelial cells.”
VEGF is a ligand secreted by many cell types including endothelial cells and raised levels are used to diagnose vasculitis.
pubmed.ncbi.nlm.nih.gov/25291956/
“VEGF levels in diagnosis of vasculitic neuropathy”
“Plasma VEGF was higher in subjects with vasculitic neuropathy as compared to controls.”
“The results from our study indicate that plasma VEGF levels are significantly associated with vasculitic neuropathy and may be used to predict this disease.”
www.ncbi.nlm.nih.gov/pmc/articles/PMC4862072/#
“Vasculitis, cerebral infarction and persistent Bartonella henselae infection in a child”
“As intravascular, intra-erythrocytic and endotheliotropic bacteria, it is possible that B. henselae initially induced a vasculitis”
www.neurology.org/doi/10.1212/NXI.0000000000000791
“A case of cerebral vasculitis due to neurobartonellosis”
To kill the vasculitis caused by Bartonella you can use your immune system and it will kill those cells and the infection inside them. This process takes time and what a person experiences depends of their level of infection.
One of the items used in this protocol is Fulvic/humic solution as it makes the checkpoint molecules more bioavailable but it also increases (NO) which helps stimulate the immune system and is a vasculitis treatment in it's own way;
www.iherb.com/pr/morningstar-minerals-inner-vitality-concentrate-fulvic-humic-minerals-2-fl-oz/65280
Bartonella’s pathogenicity can be counteracted by Nitric oxide (NO). L-arginine is the precursor to NO.
An important thing that Fulvic acid does is up-regulate NO.
pubmed.ncbi.nlm.nih.gov/18595894/
“Detrimental effects of Bartonella henselae are counteracted by L-arginine and nitric oxide in human endothelial progenitor cells”
“Nitric oxide (NO) and its precursor l-arginine (l-arg) exert a plethora of beneficial effects on vascular function and modulation of immune response. Therefore, we tested also the hypothesis that l-arg (1-30 mM) would affect the infection of B. henselae or tumor necrosis factor (TNF) in EPCs. Our data provide evidence that l-arg counteracts detrimental effects induced by TNF or Bartonella infections via NO”
www.sciencedirect.com/science/article/abs/pii/S1567576916301734
“Fulvic acid promotes extracellular anti-cancer mediators from RAW 264.7 cells, causing to cancer cell death in vitro”
“ Our data showed that FA slightly enhances cell viability in a dose-dependent manner and secretion of NO from RAW 264.7 cells. It upregulated the protein and mRNA expression of inducible NO”
pubmed.ncbi.nlm.nih.gov/22933338/
"Endothelial nitric oxide synthase inhibits the development of autoimmune-mediated vasculitis in mice"
The Fulvic/Humic solution does increase the effectiveness of the immune checkpoint protocol explained more here;
bartonella.freeforums.net/thread/113/skin-calming-detox
~d
“Every time I've tried any treatment I have worsened head pain, where my temples feel extremely sore/bruised, aching, tight constriction in every tendon or blood vessel, nerve artery, swelling protruding veins, sharp pain and its absolutely unbearable.”
I responded by saying this;
“Bartonella causes vasculitis in the whole body and when it's around the temples it's called Temporal Arteritis . There are a lot of different treatments that will affect it and when that happens the release of die off toxins cause inflammation there. The only treatment that does a complete job is the immune system but you need to turn that back on to get it to work. You also need to fix your detoxing ability to make the experience less of a burden.”
my.clevelandclinic.org/health/diseases/temporal-arteritis-giant-cell-arteritis
“Temporal arteritis, or giant cell arteritis, is a kind of vasculitis, which is inflammation of your blood vessels. With this type of vasculitis, inflammation causes your temporal arteries to swell and narrow, which restricts blood flow. The most common symptom of temporal arteritis is a throbbing headache.”
Antineutrophil cytoplasmic antibody or ANCA is an antibody that are primarily IgG. They are a type of antibody that can attach to neutrophils and cause them to attack blood vessels, which can lead to vasculitis.
This is considered an autoimmune disease but what are the neutrophils actually attacking?
pubmed.ncbi.nlm.nih.gov/34500468/
“Bartonella and Coxiella infections presenting as systemic vasculitis”
“Six cases of Bartonella infections were added to 18 cases from literature review. Causative pathogens were mainly B. henselae. Bartonella infection mimicked ANCA-associated vasculitis in 83%”
www.ncbi.nlm.nih.gov/pmc/articles/PMC6120290/
“Bartonella Endocarditis Mimicking Crescentic Glomerulonephritis with PR3-ANCA Positivity”
Obviously the heart is part of the vascular system.
pubmed.ncbi.nlm.nih.gov/38222325/
“Bartonella henselae infection-mediated shunt nephritis”
“Renal biopsy showed membranoproliferative glomerulonephritis with full-house immunofluorescence pattern. Low serum complements, PR3 ANCA positivity and elevated Bartonella immunoglobulin titers suggested a diagnosis of infective endocarditis-associated glomerulonephritis.”
Nephritis is Kidney disease.
“Is It ANCA Vasculitis or Bartonella”
When treating a Bartonella infection it might be easier to understand if you think of it as killing infected cells causing vasculitis.
pubmed.ncbi.nlm.nih.gov/11553014/#
“Evidence of a leading role for VEGF in Bartonella henselae-induced endothelial cell proliferations”
“In fact, B. henselae induces host cell production of the angiogenic factor vascular endothelial growth factor (VEGF), leading to proliferation of endothelial cells.”
VEGF is a ligand secreted by many cell types including endothelial cells and raised levels are used to diagnose vasculitis.
pubmed.ncbi.nlm.nih.gov/25291956/
“VEGF levels in diagnosis of vasculitic neuropathy”
“Plasma VEGF was higher in subjects with vasculitic neuropathy as compared to controls.”
“The results from our study indicate that plasma VEGF levels are significantly associated with vasculitic neuropathy and may be used to predict this disease.”
www.ncbi.nlm.nih.gov/pmc/articles/PMC4862072/#
“Vasculitis, cerebral infarction and persistent Bartonella henselae infection in a child”
“As intravascular, intra-erythrocytic and endotheliotropic bacteria, it is possible that B. henselae initially induced a vasculitis”
www.neurology.org/doi/10.1212/NXI.0000000000000791
“A case of cerebral vasculitis due to neurobartonellosis”
To kill the vasculitis caused by Bartonella you can use your immune system and it will kill those cells and the infection inside them. This process takes time and what a person experiences depends of their level of infection.
One of the items used in this protocol is Fulvic/humic solution as it makes the checkpoint molecules more bioavailable but it also increases (NO) which helps stimulate the immune system and is a vasculitis treatment in it's own way;
www.iherb.com/pr/morningstar-minerals-inner-vitality-concentrate-fulvic-humic-minerals-2-fl-oz/65280
Bartonella’s pathogenicity can be counteracted by Nitric oxide (NO). L-arginine is the precursor to NO.
An important thing that Fulvic acid does is up-regulate NO.
pubmed.ncbi.nlm.nih.gov/18595894/
“Detrimental effects of Bartonella henselae are counteracted by L-arginine and nitric oxide in human endothelial progenitor cells”
“Nitric oxide (NO) and its precursor l-arginine (l-arg) exert a plethora of beneficial effects on vascular function and modulation of immune response. Therefore, we tested also the hypothesis that l-arg (1-30 mM) would affect the infection of B. henselae or tumor necrosis factor (TNF) in EPCs. Our data provide evidence that l-arg counteracts detrimental effects induced by TNF or Bartonella infections via NO”
www.sciencedirect.com/science/article/abs/pii/S1567576916301734
“Fulvic acid promotes extracellular anti-cancer mediators from RAW 264.7 cells, causing to cancer cell death in vitro”
“ Our data showed that FA slightly enhances cell viability in a dose-dependent manner and secretion of NO from RAW 264.7 cells. It upregulated the protein and mRNA expression of inducible NO”
pubmed.ncbi.nlm.nih.gov/22933338/
"Endothelial nitric oxide synthase inhibits the development of autoimmune-mediated vasculitis in mice"
The Fulvic/Humic solution does increase the effectiveness of the immune checkpoint protocol explained more here;
bartonella.freeforums.net/thread/113/skin-calming-detox
~d